All larvae co-expressing psqKK111691 and EgfrUAS.cBa under the control of Scer\GAL4ap-md544 during larval stages (using Gal80[ts] for the temporal regulation of expression) wing discs tumors, associated with higher numbers of myoblasts. These phenotypes are partially suppressed by the co-expression of btlDN.UAS.
Coexpression of Rac1Scer\UAS.cLa and btlDN.Scer\UAS, under the control of Scer\GAL4ato.3.6, results in the dominance of the Rac1Scer\UAS.cLa phenotype, with a reduction in the number of dorsal cluster neuron axons crossing toward the medulla.
Coexpression of btlDN.Scer\UAS and Rac1N17.Scer\UAS, under the control of Scer\GAL4ato.3.6, results in all dorsal cluster neuron axons crossing the optic chiasm, instead of the majority retracting and branching within the lobula. This indicates that the Rac1N17.Scer\UAS phenotype is dominant.
Expression of btlDN.Scer\UAS under the control of Scer\GAL4ato.3.6 in a dsh1/+ background suppresses the reduction of dorsal cluster neuron axon extension phenotype seen in either single mutant, with the number of dorsal cluster neuron axons crossing the optic chiasm restored to almost wild-type levels.