66F5;67B1
66F;67B
66F5;67B1
66F3;67B1
Argk << bk1 << bol << l(3)67Ab << bk2 << RpS17
Df(3L)29A6/Df(3L)DocA embryos exhibit mild defects in cardioblast specification.
Df(3L)DocA/Df(3L)29A6 animals show pupal lethality.
Df(3L)29A6/+ males are fertile and have no meiotic defects.
The Df(3L)29A6 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Shows no maternal enhancement of dpphr4.
Dominantly causes tergite defects in less than 50% of run3 heterozygotes.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Homozygous embryos are very abnormal compared to wild-type.
Heterozygosity for this deletion suppresses the mutant ovarian phenotype of ovoD2.
Reduces the activity of the mitochondrial and cytoplasmic isozyme of Argk.
isolated as lethal over Df(3L)AC1
Left limit of break 1 from polytene analysis (FBrf0065574) Right limit of break 1 from inclusion of Rdl (FBrf0054082) Limits of break 2 from polytene analysis (FBrf0065574)