Approximately 0.5% of cic^fet-T6/mirr^6D1 cic^fet-U6 females exhibit a weak eggshell phenotype. The dorsal midline is expanded and the appendages are broader and positioned laterally with a trace of ventral collar material. Approximately 42.6% of cic^fet-U6/mirr^6D1 cic^fet-T6 females exhibit a moderate eggshell phenotype. The dorsal midline is expanded and the appendages are broader and positioned laterally and exhibit a ventral collar of ectopic appendage-like material. The dorsalmost region remains free of appendage material. Approximately 42.1% of cic^fet-U6/mirr^6D1 cic^fet-T6 females exhibit a moderate-severe eggshell phenotype. The dorsal midline is expanded and the appendages are broader and positioned laterally and exhibit a thick ventral collar of ectopic appendage-like material. The dorsalmost region remains free of appendage material. None of the cic^fet-U6/mirr^6D1 cic^fet-T6 females exhibit a severe eggshell phenotype (compared to 1.7% in the heterozygote). There is an increase in the weak phenotype, and a decrease in the severe phenotype compared to the cic^fet-T6/cic^fet-U6 mutant, indicating that mirr^6D1 suppresses the cic^fet-T6/cic^fet-U6 phenotype.

Stage 12 grk³/grk^2B6 ; cic^fet-U6/cic^fet-T6 double mutant egg chambers lack dorsal appendages (characteristic of loss of grk), but retain a pronounced collar of appendage material, indicating that loss of grk cannot suppress the cic phenotype.

rho⁶ mutant clones have no effect on the ectopic appendage-producing fates in cic^fet-U6/cic^fet-T6 mutant egg chambers, indicating that loss of rho function cannot suppress the cic phenotype.

Egfr^t1/Egfr^t1 ; cic^fet-T6/cic^fet-E11 females lay eggs with a dorsalised embryo (like cic^fet-T6/cic^fet-E11 single mutant females) and an eggshell that shows a combination of cic and Egfr mutant phenotypes; the dorsal appendages are broadened (as in cic mutants) but are positioned closer to the dorsal midline (as in Egfr mutants).