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General Information
Symbol
Df(1)N105
Species
D. melanogaster
Name
FlyBase ID
FBab0000593
Feature type
Computed Breakpoints include
Sequence coordinates
Member of large scale dataset(s)
Nature of Aberration
Cytological Order
Progenitor
Mutagen
Class of aberration (relative to wild type)
Causes alleles
Carries alleles
Transposon Insertions
Formalized genetic data

dy << bk1 << Flu << rad << bk2 << wy

Genetic mapping information
Comments

This deletion removes a single ribosomal protein-coding gene (though other genes are also removed).

Comments on Cytology

Ref: Craymer and Roy, 1980, D. I. S. 55: 200--204

Left limit of break 1 from polytene analysis (FBrf0034402) Right limit of break 1 from polytene analysis (FBrf0040178) Left limit of break 2 from inclusion of rad (FBrf0053305) Right limit of break 2 from polytene analysis (FBrf0034402)

Sequence Crossreferences
DNA sequence
Protein sequence
Gene Deletion and Duplication Data
Genes Deleted / Disrupted
Genes NOT Deleted / Disrupted
Genes Duplicated
Complementation Data
Completely duplicated
Partially duplicated
Molecular Data
Completely duplicated
Partially duplicated
Genes NOT Duplicated
Complementation Data
 
Molecular Data
 
Affected Genes Inferred by Location
    Phenotypic Data
    In combination with other aberrations

    Inferred to overlap with: Df(1)IE35.

    NOT in combination with other aberrations

    Flies heterozygous for the deletion do not show a Minute bristle phenotype.

    Heterozygous P1 stage pupae have a significantly increased heart rate and an increased strength of heart beat rhythmicity compared to controls.

    Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.

    No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.

    Shows no maternal enhancement of dpphr4.

    Dominantly causes tergite defects in less than 50% of run3 heterozygotes.

    Deficient embryos show an uninterpretable mutant midgut phenotype.

    Homozygous embryos are very abnormal compared to wild-type.

    Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.

    No significant change in female lethality when in combination with Sxlf1.

    Poor gut differentiation.

    Df(1)N105/+ shows a hyperkinetic phenotype.

    Stocks (2)
    Notes on Origin
    Discoverer

    Lefevre.

     
    Balancer / Genotype Variants of the Aberration
     
    Separable Components
     
    Other Comments
     

    "10F7;11D--E" was stated as revision.

    Replication normal.

    Synonyms and Secondary IDs (4)
    References (49)