Increases the frequency of the trx bithorax-variegated phenotype in heterozygous combination with Df(3R)red-P52.
Lethal in heterozygous combination with Tp(3;1)P115. Lethal in heterozygous combination with Df(3R)P115. Lethal in heterozygous combination with Df(3R)P115 Dp(3;3)S462. Lethal in heterozygous combination with Df(3R)P10. Lethal in heterozygous combination with Df(3R)P10 Dp(3;3)sbd104. Lethal in heterozygous combination with Df(3R)Ubx109. Viable in heterozygous combination with Tp(3;2)iab2P10. Viable in heterozygous combination with Tp(3;3)sbd104. Lethal in heterozygous combination with Df(3R)sbd104. Lethal in heterozygous combination with Tp(3;3)bxd100.
Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2.
viable
Homozygous lethal.
The Df(3R)bxd100 chromosome may act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but the eye colour phenotype in the presence of Df(3R)bxd100 overlaps the eye colour phenotype in a wild-type background so it cannot be unequivocally demonstrated that the deficiency chromosome uncovers a suppressor of telomeric silencing. In addition, any suppression is a false positive result (the suppressor is not within the bounds of the deficient region) because the region of the deficiency is covered by one or more nonsuppressing deficiencies.