Low-frequency RNA-Seq exon junction(s) not annotated.
Gene model reviewed during 5.53
There is only one protein coding transcript and one polypeptide associated with this gene
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\cv-d using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\cv-d in GBrowse 2
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Shows particularly robust cycling of transcription in adult heads, as assessed by expression analysis using high density oligonucleotide arrays with probe generated during three 12-point time course experiments over the course of 6 days. Shows significant change of expression pattern in circadian mutant background; decreased expression in per01, tim01 and increased expression in ClkJrk background.
Mutations in cv-d remove transverse veins or crossveins: crossvein specific. All anterior and posterior crossveins are removed leaving the sensilla campaniformia of the anterior crossvein. Slightly abnormal tarsal joints in the leg. Shows additive interactions with vn, ve, ci, cg, shv, ast, H, Vno, vvl, ri, tg, tt and ab.
ri, tg, tt, ab, cv, cv-2, cv-c and cv-d belong to the radius incompletus phenotypic group within the 'lack-of-vein' mutant class. Loss-of-function alleles at these loci remove stretches of veins in two or more longitudinal veins. Double mutants of this group have additive phenotypes, suggesting the genes are vein-specifi, and small lanceolate wings. Genes are therefore involved in whole vein-region specification rather than vein differentiation. ab and cv-d mutants have a pleiotropic effect on leg development.
Duncan, March 1934.