Co-expression of da^Scer\UAS.cGa results in increased size of ectopic eyes induced by Scer\GAL4^30A>ey^Scer\UAS.cHa.

Expression of da^Scer\UAS.cGa under the control of Scer\GAL4^tub.PU in clones in the wing disc still induces the formation of sensory organ precursor cells in a Df(1)260-1 background, although at lower frequency than in a wild-type background.

Coexpression of both da^Scer\UAS.cGa and sens^Scer\UAS.cNa, under the control of Scer\GAL4^sca-109-68, generates 63.5 sensory organs, composed of both bristles and campaniform sensilla, along the third wing vein. This is an enhancement of the phenotype seen when either transgene is expressed alone and is greater than an additive effect.

Expression of da^Scer\UAS.cGa in the thorax, under the control of Scer\GAL4^Eq1, leads to the generation of numerous bristles on the thorax of sc^10-1 mutants. These mutants have no thoracic bristles without the transgene expression.

Coexpression of da^Scer\UAS.cGa and sens^Scer\UAS.cNa in the thorax, under the control of Scer\GAL4^Eq1, leads to the generation of multiple bristles on the thorax of sc^10-1 mutants; these bristles are present at a much greater density than those on wild-type thoraces or indeed the density produced by expressing either da^Scer\UAS.cGa or sens^Scer\UAS.cNa alone.

Overexpression of da^Scer\UAS.cGa, under the control of Scer\GAL4^Eq1, results in the formation of bristles in Df(1)260-1 clones, which are usually devoid of bristles.

Co-expression of da^Scer\UAS.cGa and nvy^Scer\UAS.cWa, both under the control of Scer\GAL4^pnr-MD237, causes a bristle phenotype that is intermediate to the two phenotypes produced by single expression of either; flies have fewer macrochaetae on the thorax than wild type. Inhibition of nvy expression, achieved by expressing nvy^{dsRNA.Scer\UAS} under the control of Scer\GAL4^pnr-MD237, enhances the extra bristle phenotype of Scer\GAL4^pnr-MD237 > da^Scer\UAS.cGa flies.

Co-expression of da^Scer\UAS.cGa does not significantly alter the small eye phenotype caused by expression of wg^{wg.3'UTR.Scer\UAS} under the control of Scer\GAL4^GMR.PF, but does fully suppress the loss of interommatidial bristles seen in these flies. Co-expression of da^Scer\UAS.cGa restores some progression of the morphogenetic furrow in animals expressing wg^{wg.3'UTR.Scer\UAS} under the control of Scer\GAL4^dpp.blk1. Co-expression of da^Scer\UAS.cGa significantly rescues progression of the morphogenetic furrow in animals expressing arm^{S10.Scer\UAS.T:Hsap\MYC} under the control of Scer\GAL4^dpp.blk1.

When twi¹/+ is added to da^Scer\UAS.cGa, Scer\GAL4^twi.PB embryos, there is a dramatic suppression of somatic muscle fate. No increase in other mesodermal tissues is seen.

When twi¹/+ is added to da^Scer\UAS.cGa, Scer\GAL4^twi.PB embryos, a dramatic suppression of somatic muscle fate. No increase in other mesodermal tissues is seen.

Expression of both amos^Scer\UAS.cHa and da^Scer\UAS.cGa under the control of Scer\GAL4^sca-537.4 results in the induction of numerous elav-positive neurons.

Co-expression of l(1)sc^Scer\UAS.cHa and da^Scer\UAS.cGa driven by Scer\GAL4^da.G32 results in muscle and PNS pattern formation defects and in very rare cases, muscle duplication. The addition of l(1)sc^Scer\UAS.cHa and da^Scer\UAS.cGa does not rescue the mesoderm phenotype of N^{int.SH.Scer\UAS} driven by Scer\GAL4^da.G32.

Some embryos coexpressing l(1)sc^Scer\UAS.cHa and da^Scer\UAS.cGa under the control of Scer\GAL4^da.G32 develop multiple Malpighian tubule tip cells.

Coexpression of da^Scer\UAS.cGa with l(1)sc^Scer\UAS.cHa causes a similar phenotype to Scer\GAL4^da.G32-mediated expression of da^Scer\UAS.cGa. Scer\GAL4^da.G32-mediated expression of Dl::N^{ΔECN.Scer\UAS} generates aneural embryos. Simultaneous expression of da^Scer\UAS.cGa and l(1)sc^Scer\UAS.cHa does not rescue the phenotype.