Expression of InR^GD104 under the control of Scer\GAL4^Orco.PU or Scer\GAL4^Or47a.PU does not alter the starvation-dependent increase in attraction to pentyl acetate or the levels of food intake as compared to control larvae. It leads to a significant increase in body weight under the control of Scer\GAL4^Orco.PU when compared to control larvae.

The adulthood-only expression of InR^GD104 under the combined control of Scer\GAL4^C587 and Gal80[ts] leads to several germarium defects compared to controls, including: smaller germaria with fewer cysts and budding fewer egg chambers; a significant and progressive decrease in the number of escort cells, which fail to completely wrap germ cell cysts; germline stem cells dividing less frequently and progressively decreasing in number; and germ cell cysts showing a lower proportion of cell division; but no defect in the number of cap cells.

The adulthood-only expression of InR^GD104 leads to a significant and progressive decrease in the number of germline stem cells in the adult germarium when under the control of Scer\GAL4^ptc-559.1, while leading to a significant decrease in the number of escort cells in the adult germarium when under the control of Scer\GAL4^bab1-Gal4, as compared to controls; the Scer\GAL4^bab1-Gal4-driven expression also leads to a significant decrease in the number of cap cells in the adult germarium, as compared to controls.

Escort cell (within clones) expressing InR^GD104 under the control of Scer\GAL4^Act5C.PU show blunted extension of membrane processes that fail to completely wrap germ cell cysts, as compared to controls.

Presynaptic expression of InR^GD104 under the control of Scer\GAL4^elav-C155 causes synaptic retractions in less than 10% of synapses.

Expression of InR^GD104 in the posterior wing compartment under the control of Scer\GAL4^en-e16E does not affect wing vein formation.

Adults expressing InR^GD104 under the control of Scer\GAL4^elav.PLu (in the presence of Dcr-2^Scer\UAS.cDa to increase the efficiency of RNAi) do not show a significant defect in avoidance of noxious temperature (46[o]C) compared to control flies.

Depending on the insertion line used, expression under the control of Scer\GAL4^Mef2.PR can result in a weak flyer phenotype or early pupal lethality.

Expression under the control of Scer\GAL4^pnr-MD237 may result in semi-lethality, depending on the insertion line used.

Expression under the control of Scer\GAL4^pnr-MD237 results in bristle morphology defects on the notum in 100% or 80-90% of the Scer\GAL4^pnr-MD237 expression domain, depending on the insertion line used.

Expression of InR^GD104 under the control of Scer\GAL4^Act during the pupal stages (Scer\GAL80^ts.αTub84B present in the animals has been inactivated by shifting to the restrictive temperature following the larval/pupal transition) results in smaller and lighter adults than normal, with reduced appendages.

InR^GD104, Scer\GAL4^elav-C155 has abnormal starvation stress response | adult stage phenotype, non-enhanceable by mir-137^CR

InR^GD104, Scer\GAL4^elav-C155 has abnormal starvation stress response | adult stage phenotype, non-enhanceable by mir-137^KO

Scer\GAL4^elav-C155/InR^GD104 is a non-enhancer of abnormal starvation stress response | adult stage | semidominant phenotype of mir-137^CR

Scer\GAL4^elav-C155/InR^GD104 is a non-enhancer of abnormal starvation stress response | adult stage | semidominant phenotype of mir-137^KO

Scer\GAL80^ts.αTub84B, InR^GD104, Scer\GAL4^da.PU is a non-enhancer of lethal - all die during pupal stage | heat sensitive phenotype of Scer\GAL4^da.PU, Scer\GAL80^ts.αTub84B, fzr^RNAi.UAS.WIZ

InR^GD104, Scer\GAL4^esg.PU is a suppressor of increased cell number phenotype of Scer\GAL4^esg.PU, mir-305^{sponge.UAS.DsRed(Unk)}

InR^GD104, Scer\GAL4^{FRT.Rnor\Cd2.Act5C} is a suppressor of neoplasia phenotype of Ras85D^V12.UAS, Scer\GAL4^{FRT.Rnor\Cd2.Act5C}, dlg1^GD4689

InR^GD104, Scer\GAL4^esg.PU is a suppressor of intestinal stem cell | adult stage phenotype of Scer\GAL4^esg.PU, mir-305^{sponge.UAS.DsRed(Unk)}

InR^GD104, Scer\GAL4^esg.PU is a suppressor of enteroblast | adult stage phenotype of Scer\GAL4^esg.PU, mir-305^{sponge.UAS.DsRed(Unk)}