oho23B, l(2)168/14, l(2)k16814, oho23, overgrown hematopoietic organs at 23B
Low-frequency RNA-Seq exon junction(s) not annotated.
Annotated transcripts do not represent all supported alternative splices within 5' UTR.
Gene model reviewed during 5.47
0.7, 0.4 (northern blot)
83 (aa); 9.3 (kD predicted)
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\RpS21 using the Feature Mapper tool.
GBrowse - Visual display of RNA-Seq signalsView Dmel\RpS21 in GBrowse 2
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for identity of: RpS21 oho23B
Deletions removing oho23B but no other cytoplasmic ribosomal protein-encoding genes show Minute phenotypes.
oho23B acts as a translation initiation factor, rather than as a core ribosomal protein.
Mutants isolated in a screen of the second chromosome identifying genes affecting disc morphology.
The tumour suppressor gene oho23B encodes a homologue of the human S21 ribosomal protein.
Mutants display a blood cell neoplastic phenotype.
Genetic and developmental characteristics of the lymph gland overgrowth mutant have been determined.