l(1)ESHS36 << bk1 << eggroll << l(1)7Do << bk2 << rdgA
Breakpoint(s) molecularly mapped
The viability of Df(1)svr/Df(1)RA2 or Df(1)svr/+ female progeny derived from a cross between Df(1)RA2/+ females and Df(1)svr/Y males is severely reduced. The viability of Df(1)N71/Df(1)RA2 or Df(1)N71/+ female progeny derived from a cross between Df(1)RA2/+ females and Df(1)N71/Y males is severely reduced.
Df(1)RA2/Ubl females slight oc phenotype
Does not cause unconditional lethality in hybrid females when heterozygous with D.simulans chromosome.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Homozygous embryos are very abnormal compared to wild-type.
Heterozygosity for this deletion enhances the mutant ovarian phenotype of ovoD2.
More than six-fold reduction in sex ratio and significant levels of female lethality is observed when in combination with Sxlf1. Female lethality is also reduced when in combination with da1 but restored when in combination with emcML.
The viability of Sxlf1/Df(1)RA2 female progeny derived from a cross between Df(1)RA2/+ females and Sxlf1/Y males is severely reduced. Viability is recovered if the Sxlf1/Df(1)RA2 female progeny also carry Dp(1;3)sn13a1 or if the Df(1)RA2/+ mothers also carry Dp(1;3)sn13a1.
Heterozygotes have ocelli placed far back on the head and there is a slight indentation in the cuticle beneath the postvertical bristles.