wing & macrochaeta
wing vein L3 & sensillum campaniformium
Heterozygous bs03267 flies have small patches of ectopic vein in the vicininty of L2 and L5. If these flies are also homozygous for ash2S112411 their wings are small, blistered and exhibit localised reduction and loss of intervein betwee L2 and L3 and between L4 and L5 and ectopic crossveins. Fusions occur between L2 and L3 and between L4 and L5, particularly proximally.
Dominantly enhances the frequency of br1 mutant animals with malformed legs. This phenotype is cold-sensitive; at 18oC, 73% of br1/Y ; bs03267/+ animals have malformed legs, at 21oC, the frequency of malformed legs is 15%.
Hemizygosity for bs totally suppresses the lack of vein L4 phenotype of loss of function Egfr, rl, vn and rho mutants and greatly enhances the amount of ectopic vein in rlSem and rhohs.PSt, even in the absence of heat shock. Double mutant wing clones with Egfrf37 occupy vein territories like those for bs03267 but with a frequency and size similar to Egfrf37 controls. Double mutant cells differentiate autonomously into pigmented, corrugated and compacted cells characteristic of the vein histotype. bs03267/bs2; rho9, vnM2/rhove-1, vn1 triple mutants show an almost wild type wing vein pattern.