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General Information
Symbol
Dmel\KrIf-1
Species
D. melanogaster
Name
FlyBase ID
FBal0005583
Feature type
allele
Associated gene
Associated Insertion(s)
Carried in Construct
Also Known As
If
Key Links
Mutagen
Nature of the Allele
Mutations Mapped to the Genome
 
Type
Location
Additional Notes
References
Associated Sequence Data
DNA sequence
Protein sequence
 
 
Progenitor genotype
Cytology
Nature of the lesion
Statement
Reference
Expression Data
Reporter Expression
Additional Information
Statement
Reference
 
Marker for
Reflects expression of
Reporter construct used in assay
Human Disease Associations
Disease Ontology (DO) Annotations
Models Based on Experimental Evidence ( 0 )
Disease
Evidence
References
Modifiers Based on Experimental Evidence ( 0 )
Disease
Interaction
References
Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
 
Disease-implicated variant(s)
 
Phenotypic Data
Phenotypic Class
Phenotype Manifest In
Detailed Description
Statement
Reference

Mutants have an eye outgrowth phenotype.

Feeding geldanamycin to homozygous females increases the frequency of the ectopic eye outgrowth phenotype by approximately 10-fold.

KrIf-1/+ flies have small rough eyes.

The size of the eye is reduced and the ommatidia ared fused or absent in the ventral part of the eye and irregularly arranged dorsally.

Heterozygotes have small, narrow eyes that are pointed ventrally. In the ventral portion of the eye the facets are fused or absent and dorsally they are irregularly arranged. Eyes are further reduced in size in homozygotes, forming narrow slits with a glossy surface lacking most ommatidia.

The number of ommatidia is abnormal in heterozygotes.

Displays locomotor activity rhythm with an approximately 24h period.

KrIf-1 males that are also mutant for mle4 show a more severe phenotype than their mle4/+ sibs.

KrIf-1 results in a rough eye phenotype which is more extreme in homozygotes. Eye discs are deeply folded and smaller than wild type. Ommatidia begin to form posterior to the morphogenetic furrow but later stages of ommatidial development are more rarely seen. The adult eye is greatly reduced in size and is made up of pigment cells and lacunae. Photoreceptor rhabdomeres are only rarely found, and have unusual morphology. The eye region sometimes differentiates cuticle.

The mutant phenotype of KrIf-1 is due to a developmental defect in precursor cells. When heterozygous with apx1 the adult eye is half the normal size, the ventral half more prone to irregularities. Eye disc has a fairly large number of clusters with irregular arrangements. When heterozygous with st adult eye is narrow and ventral half is devoid of facets. Rhabdomeres are fused and only 20 ommatidia are present. Eye disc size is reduced with few clusters.

The eye area is reduced to approximately one-half that of wild-type in heterozygotes, it is oblong and generally pointed ventrally. The facets are irregularly distributed, are frequently missing across the middle of the eye, and are sometimes fused or absent in the ventral portion. The eye is a narrow slit in homozygotes and has a smooth glossy surface. Viability and fecundity of both hetero- and homozygotes is good to excellent.

In heterozygote, eye area about one-half normal; narrow and pointed ventrally; facets irregular and often missing across middle of eyes, sometimes fused or absent in ventral portion. In homozygote, eyes are narrow slits with smooth glossy surface. In the eye disk of late third instar larvae, fairly large number of cell clusters in irregular arrangement, especially in ventral half of disk (Renfranz and Benzer, 1989). Viability and fertility good. RK1.

External Data
Interactions
Show genetic interaction network for Enhancers & Suppressors
Phenotypic Class
Enhanced by
Statement
Reference

KrIf-1 has visible phenotype, enhanceable by piwi[+]/piwi1

KrIf-1 has visible phenotype, enhanceable by piwi[+]/piwi2

KrIf-1 has visible phenotype, enhanceable by Hop[+]/Stip1k00616

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2L)E55

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)Px2

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)bw-S46

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)bw5

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)cn9

KrIf-1 has visible | dominant phenotype, enhanceable by GstS104227

KrIf-1 has visible | dominant phenotype, enhanceable by In(2LR)Px4

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)pk78s

KrIf-1 has visible | dominant phenotype, enhanceable by Kr-h110642

KrIf-1 has visible | dominant phenotype, enhanceable by Mi-21

KrIf-1 has visible | dominant phenotype, enhanceable by Rab608323

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2L)Prl

KrIf-1 has visible | dominant phenotype, enhanceable by S05671

KrIf-1 has visible | dominant phenotype, enhanceable by S07056

KrIf-1 has visible | dominant phenotype, enhanceable by Sema2a03021

KrIf-1 has visible | dominant phenotype, enhanceable by crol10639

KrIf-1 has visible | dominant phenotype, enhanceable by dpp10638

KrIf-1 has visible | dominant phenotype, enhanceable by ex01270

KrIf-1 has visible | dominant phenotype, enhanceable by ex02278

KrIf-1 has visible | dominant phenotype, enhanceable by CCT803996

KrIf-1 has visible | dominant phenotype, enhanceable by chinmo04111

KrIf-1 has visible | dominant phenotype, enhanceable by l(2)0454804548

KrIf-1 has visible | dominant phenotype, enhanceable by Df(2R)PC4

KrIf-1 has visible | dominant phenotype, enhanceable by l(2)0849208492

KrIf-1 has visible | dominant phenotype, enhanceable by osa00090

KrIf-1 has visible | dominant phenotype, enhanceable by styΔ5

KrIf-1 has visible | dominant phenotype, enhanceable by Mi-21/Mi-2[+]

KrIf-1 has visible | dominant phenotype, enhanceable by osa00090/osa[+]

KrIf-1 has visible | dominant phenotype, enhanceable by Fer2LCH[+]/Fer2LCH07016

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)brm11

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)P115

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)kar-D3

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)GB104

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3R)p712

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)R-G7

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)fz-GF3b

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)W10

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)W4

KrIf-1 has visible | dominant phenotype, enhanceable by Df(3L)kto2

KrIf-1 has visible | dominant phenotype, enhanceable by osa[+]/osa04539

Suppressed by
Statement
Reference

KrIf-1 has visible | dominant phenotype, suppressible by Df(2R)trix

KrIf-1 has visible | dominant phenotype, suppressible by Sc205634

KrIf-1 has visible | dominant phenotype, suppressible by Vha55j2E9

KrIf-1 has visible | dominant phenotype, suppressible by bun05479

KrIf-1 has visible | dominant phenotype, suppressible by bun06475

KrIf-1 has visible | dominant phenotype, suppressible by caps02937

KrIf-1 has visible | dominant phenotype, suppressible by emc05592

KrIf-1 has visible | dominant phenotype, suppressible by styL1

KrIf-1 has visible | dominant phenotype, suppressible by bun02687

KrIf-1 has visible | dominant phenotype, suppressible by Eip75B07041

KrIf-1 has visible | dominant phenotype, suppressible by In(2L)05887

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)Ar12-1

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)Ar14-8

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)GN19

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)M21

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)HR119

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)AC1

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)Ly

KrIf-1 has visible | dominant phenotype, suppressible by Df(3L)H99

KrIf-1 has visible | dominant phenotype, suppressible by Df(3R)M-Kx1

KrIf-1 has visible | dominant phenotype, suppressible by l(3)06240[+]/Psa06240

KrIf-1 has visible | dominant phenotype, suppressible by l(3)05967[+]/l(3)0596705967

KrIf-1 has visible | dominant phenotype, suppressible by l(3)neo3[+]/l(3)neo31

KrIf-1 has visible | dominant phenotype, suppressible by emc[+]/emc05592

KrIf-1 has visible | dominant phenotype, suppressible by Sc2[+]/Sc205634

KrIf-1 has visible | dominant phenotype, suppressible by Sap13006924/l(3)06924[+]

KrIf-1 has visible | dominant phenotype, suppressible by caps02937/caps[+]

KrIf-1 has visible | dominant phenotype, suppressible by Eip75B[+]/Eip75B07041

KrIf-1 has visible | dominant phenotype, suppressible by rept[+]/rept06945

KrIf-1 has visible | dominant phenotype, suppressible by Vha55j2E9/Vha55[+]

KrIf-1 has visible | dominant phenotype, suppressible by l(3)j2C3[+]/l(3)j2C3j2C3

KrIf-1 has visible | dominant phenotype, suppressible by l(3)neo391/l(3)neo39[+]

KrIf-1 has visible | dominant phenotype, suppressible by l(3)neo47[+]/l(3)neo471

Phenotype Manifest In
Enhanced by
Statement
Reference

KrIf-1 has eye phenotype, enhanceable by piwi[+]/piwi1

KrIf-1 has eye phenotype, enhanceable by piwi[+]/piwi2

KrIf-1 has eye phenotype, enhanceable by Hop[+]/Stip1k00616

KrIf-1 has eye phenotype, enhanceable by Df(2R)Px2

KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)Px2

KrIf-1 has eye phenotype, enhanceable by Df(2R)bw-S46

KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)bw-S46

KrIf-1 has eye phenotype, enhanceable by Df(2R)bw5

KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)bw5

KrIf-1 has eye phenotype, enhanceable by Df(2L)E55

KrIf-1 has eye phenotype, enhanceable by Df(2R)cn9

KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)cn9

KrIf-1 has eye phenotype, enhanceable by GstS104227

KrIf-1 has ommatidium phenotype, enhanceable by GstS104227

KrIf-1 has eye phenotype, enhanceable by In(2LR)Px4

KrIf-1 has ommatidium phenotype, enhanceable by In(2LR)Px4

KrIf-1 has eye phenotype, enhanceable by Df(2R)pk78s

KrIf-1 has ommatidium phenotype, enhanceable by Df(2L)E55

KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)pk78s

KrIf-1 has eye phenotype, enhanceable by Kr-h110642

KrIf-1 has ommatidium phenotype, enhanceable by Kr-h110642

KrIf-1 has eye phenotype, enhanceable by Mi-21

KrIf-1 has ommatidium phenotype, enhanceable by Mi-21

KrIf-1 has eye phenotype, enhanceable by Rab608323

KrIf-1 has ommatidium phenotype, enhanceable by Rab608323

KrIf-1 has eye phenotype, enhanceable by S05671

KrIf-1 has ommatidium phenotype, enhanceable by S05671

KrIf-1 has eye phenotype, enhanceable by S07056

KrIf-1 has ommatidium phenotype, enhanceable by S07056

KrIf-1 has eye phenotype, enhanceable by Sema2a03021

KrIf-1 has ommatidium phenotype, enhanceable by Sema2a03021

KrIf-1 has eye phenotype, enhanceable by Df(2L)Prl

KrIf-1 has eye phenotype, enhanceable by crol10639

KrIf-1 has ommatidium phenotype, enhanceable by crol10639

KrIf-1 has eye phenotype, enhanceable by dpp10638

KrIf-1 has ommatidium phenotype, enhanceable by dpp10638

KrIf-1 has eye phenotype, enhanceable by ex01270

KrIf-1 has ommatidium phenotype, enhanceable by ex01270

KrIf-1 has eye phenotype, enhanceable by ex02278

KrIf-1 has ommatidium phenotype, enhanceable by Df(2L)Prl

KrIf-1 has ommatidium phenotype, enhanceable by ex02278

KrIf-1 has eye phenotype, enhanceable by CCT803996

KrIf-1 has ommatidium phenotype, enhanceable by CCT803996

KrIf-1 has eye phenotype, enhanceable by chinmo04111

KrIf-1 has ommatidium phenotype, enhanceable by chinmo04111

KrIf-1 has eye phenotype, enhanceable by l(2)0454804548

KrIf-1 has ommatidium phenotype, enhanceable by l(2)0454804548

KrIf-1 has eye phenotype, enhanceable by l(2)0849208492

KrIf-1 has ommatidium phenotype, enhanceable by l(2)0849208492

KrIf-1 has eye phenotype, enhanceable by osa00090

KrIf-1 has ommatidium phenotype, enhanceable by osa00090

KrIf-1 has eye phenotype, enhanceable by styΔ5

KrIf-1 has ommatidium phenotype, enhanceable by styΔ5

KrIf-1 has eye phenotype, enhanceable by Df(2R)PC4

KrIf-1 has ommatidium phenotype, enhanceable by Df(2R)PC4

KrIf-1 has eye phenotype, enhanceable by osa[+]/osa04539

KrIf-1 has eye phenotype, enhanceable by Mi-21/Mi-2[+]

KrIf-1 has eye phenotype, enhanceable by osa00090/osa[+]

KrIf-1 has eye phenotype, enhanceable by Fer2LCH[+]/Fer2LCH07016

KrIf-1 has eye phenotype, enhanceable by Df(3L)brm11

KrIf-1 has eye phenotype, enhanceable by Df(3R)P115

KrIf-1 has eye phenotype, enhanceable by Df(3R)kar-D3

KrIf-1 has eye phenotype, enhanceable by Df(3R)GB104

KrIf-1 has eye phenotype, enhanceable by Df(3R)p712

KrIf-1 has eye phenotype, enhanceable by Df(3L)R-G7

KrIf-1 has eye phenotype, enhanceable by Df(3L)fz-GF3b

KrIf-1 has eye phenotype, enhanceable by Df(3L)W10

KrIf-1 has eye phenotype, enhanceable by Df(3L)W4

KrIf-1 has eye phenotype, enhanceable by Df(3L)kto2

NOT Enhanced by
Statement
Reference

KrIf-1 has eye phenotype, non-enhanceable by Low1

Suppressed by
Statement
Reference

KrIf-1 has eye phenotype, suppressible by Sc205634

KrIf-1 has ommatidium phenotype, suppressible by Sc205634

KrIf-1 has eye phenotype, suppressible by Vha55j2E9

KrIf-1 has ommatidium phenotype, suppressible by Vha55j2E9

KrIf-1 has eye phenotype, suppressible by bun05479

KrIf-1 has ommatidium phenotype, suppressible by bun05479

KrIf-1 has eye phenotype, suppressible by Df(2R)trix

KrIf-1 has eye phenotype, suppressible by bun06475

KrIf-1 has ommatidium phenotype, suppressible by bun06475

KrIf-1 has eye phenotype, suppressible by caps02937

KrIf-1 has ommatidium phenotype, suppressible by caps02937

KrIf-1 has eye phenotype, suppressible by emc05592

KrIf-1 has ommatidium phenotype, suppressible by emc05592

KrIf-1 has eye phenotype, suppressible by styL1

KrIf-1 has ommatidium phenotype, suppressible by Df(2R)trix

KrIf-1 has ommatidium phenotype, suppressible by styL1

KrIf-1 has eye phenotype, suppressible by bun02687

KrIf-1 has ommatidium phenotype, suppressible by bun02687

KrIf-1 has eye phenotype, suppressible by Eip75B07041

KrIf-1 has ommatidium phenotype, suppressible by Eip75B07041

KrIf-1 has eye phenotype, suppressible by In(2L)05887

KrIf-1 has ommatidium phenotype, suppressible by In(2L)05887

KrIf-1 has eye phenotype, suppressible by Df(3L)Ar12-1

KrIf-1 has eye phenotype, suppressible by Df(3L)Ar14-8

KrIf-1 has eye phenotype, suppressible by Df(3L)GN19

KrIf-1 has eye phenotype, suppressible by Df(3L)M21

KrIf-1 has eye phenotype, suppressible by Df(3L)HR119

KrIf-1 has eye phenotype, suppressible by Df(3L)AC1

KrIf-1 has eye phenotype, suppressible by Df(3L)Ly

KrIf-1 has eye phenotype, suppressible by Df(3L)H99

KrIf-1 has eye phenotype, suppressible by Df(3R)M-Kx1

KrIf-1 has eye phenotype, suppressible by l(3)06240[+]/Psa06240

KrIf-1 has eye phenotype, suppressible by l(3)05967[+]/l(3)0596705967

KrIf-1 has eye phenotype, suppressible by l(3)neo3[+]/l(3)neo31

KrIf-1 has eye phenotype, suppressible by emc[+]/emc05592

KrIf-1 has eye phenotype, suppressible by Sc2[+]/Sc205634

KrIf-1 has eye phenotype, suppressible by Sap13006924/l(3)06924[+]

KrIf-1 has eye phenotype, suppressible by caps02937/caps[+]

KrIf-1 has eye phenotype, suppressible by Eip75B[+]/Eip75B07041

KrIf-1 has eye phenotype, suppressible by rept[+]/rept06945

KrIf-1 has eye phenotype, suppressible by Vha55j2E9/Vha55[+]

KrIf-1 has eye phenotype, suppressible by l(3)j2C3[+]/l(3)j2C3j2C3

KrIf-1 has eye phenotype, suppressible by l(3)neo391/l(3)neo39[+]

KrIf-1 has eye phenotype, suppressible by l(3)neo47[+]/l(3)neo471

KrIf-1 has eye phenotype, suppressible by exba[+]/kra03022

NOT suppressed by
Statement
Reference

KrIf-1 has eye phenotype, non-suppressible by Low1

Enhancer of
Statement
Reference

KrIf-1/Kr[+] is an enhancer of eye phenotype of Scer\GAL4GMR.PF, fruNP0021

Other
Statement
Reference

KrIf-1, kto[+]/kto1 has eye phenotype

KrIf-1, osa2/osa[+] has eye phenotype

KrIf-1, skd2/skd[+] has eye phenotype

KrIf-1, Nsf2urd-2/urd[+] has eye phenotype

KrIf-1, vtd[+]/vtd3 has eye phenotype

KrIf-1, z[+]/zv77h has eye phenotype

Hsp83e4A/Hsp83[+], KrIf-1 has eye phenotype

Hsp83[+]/Hsp83e6A, KrIf-1 has eye phenotype

Hsp83e6D/Hsp83[+], KrIf-1 has eye phenotype

KrIf-1, Trl[+]/TrlR85 has eye phenotype

KrIf-1, brm[+]/brm2 has eye phenotype

KrIf-1, btldev2/btl[+] has eye phenotype

KrIf-1, fs(1)h1/fs(1)h[+] has eye phenotype

Additional Comments
Genetic Interactions
Statement
Reference

piwi1/+ and piwi2/+ enhance the eye outgrowth phenotype phenotype of KrIf-1; approximately 7% of the progeny have an eye outgrowth phenotype in a cross of piwi1/+ or piwi2/+ females to KrIf-1/+ males. However, the reciprocal cross of KrIf-1/+ females to piwi1/+ or piwi2/+ males does not result in progeny with an eye outgrowth phenotype, indicating a maternal effect. In addition, the expression of the eye outgrowth phenotype depends on the presence of the piwi2 mutation in the progeny, indicating a zygotic effect.

The ability of geldanamycin to increase the frequency of the ectopic eye outgrowth phenotype in KrIf-1 homozygous females is reduced if the females are also carrying piwi5'T:Hsap\MYC.

Maternal Hopk00616 is a dominant enhancer of the eye outgrowth phenotype phenotype of KrIf-1.

A KrIf-1 background strongly enhances the rough eye phenotype found upon expression of fruNP0021 under the control of Scer\GAL4GMR.PF.

15+/-4% of KrIf-1/+ flies born to Hsp83e1D/+ mothers have outgrowths with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

11+/-3% of KrIf-1/+ flies born to Hsp83e3A/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

6+/-2% of KrIf-1/+ flies born to Hsp83e4A/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

6+/-2% of KrIf-1/+ flies born to Hsp83e6A/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

13+/-4% of KrIf-1/+ flies born to Hsp83e6D/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

5+/-1% of KrIf-1/+ flies born to brm2/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

10+/-2% of KrIf-1/+ flies born to fs(1)h1/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

15+/-4% of KrIf-1/+ flies born to kto1/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

10+/-2% of KrIf-1/+ flies born to osa2/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

2+/-1% of KrIf-1/+ flies born to skd2/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

5+/-2% of KrIf-1/+ flies born to urd2/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

55+/-8% of KrIf-1/+ flies born to vtd3/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

5+/-2% of KrIf-1/+ flies born to zv77h/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

10+/-3% of KrIf-1/+ flies born to btldev2/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

10+/-2% of KrIf-1/+ flies born to TrlR85/+ mothers have outgrowths

with ectopic vibrissae protruding from the ventral region of the eye,

compared to less than 0.1% of KrIf-1/+ flies born to isogenised

wild-type mothers.

KrIf-1, ex01270 flies develop smaller eyes and fewer ommatidia than KrIf-1 flies. KrIf-1, S05671 eyes show a severe enhancement of the KrIf-1 phenotype; the eyes only have only remnants of a few ommatidia.

The eye phenotype is unaffected by Low1.

Xenogenetic Interactions
Statement
Reference
Complementation and Rescue Data
Comments
Images (0)
Mutant
Wild-type
Stocks (105)
Notes on Origin
Discoverer

Casey, 16th Dec. 1965.

Casey.

External Crossreferences and Linkouts ( 0 )
Synonyms and Secondary IDs (5)
References (17)