bk1 hits noc << Adh << bk2 << mol
Lesion within noc.
Cytologically invisible deletion.
Left limit of break 1 from polytene analysis (FBrf0038047) Right limit of break 1 from inclusion of Adh (FBrf0038047) Limits of break 2 from polytene analysis (FBrf0038047)
Df(2L)A178 complements mutations in the l(2)35Ba region of noc but does not complement the nocA region of noc.
Weakly suppresses the zeste phenotype in a z1 w11E4; Tp(1;2)TE35B homologue.
Chromosome acts as a weak trans-suppressor of a physical interaction between tandemly duplicated copies of w+, an interaction that is required for the suppression of genes by z1.
Semilethal with noc. Males and females homozygous viable and fertile with strong noc and osp phenotypes
selected as ADH null on 1-penten-3-ol. Induced with, but genetically separable from T(2;3)CA3.
Reported to delete Adhr in error; see FBrf0141768 for correction.
nLA178 chromosome carries an independent translocation, T(2;3)CA3, that can be removed by exchange.