BG:DS09218.1 , Dbf4, BG:DS09218.2
Please see the JBrowse view of Dmel\chif for information on other features
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Multiphase exon postulated: this gene shares a region of coding sequence with an overlapping gene, but different reading frames are utilized in the overlapping coding region; overlap >20aa.
Gene model reviewed during 5.43
Gene model reviewed during 5.50
Complex/atypical overlap: Shares 5' UTR with downstream gene; in addition, one transcript isoform shares 3' UTR with downstream gene.
Gene model reviewed during 6.27
6.6, 6.5 (northern blot)
1711, 1695 (aa)
Click to get a list of regulatory features (enhancers, TFBS, etc.) and gene disruptions (point mutations, indels, etc.) within or overlapping Dmel\chif using the Feature Mapper tool.
Comment: anlage in statu nascendi
Comment: anlage in statu nascendi
Comment: reported as procephalic ectoderm anlage
Comment: reported as procephalic ectoderm anlage
Comment: reported as procephalic ectoderm anlage
Comment: reported as procephalic ectoderm anlage
Comment: reported as procephalic ectoderm primordium
Comment: reported as procephalic ectoderm primordium
Comment: reported as procephalic ectoderm primordium
Comment: reported as procephalic ectoderm primordium
Comment: reported as procephalic ectoderm primordium
Comment: reported as procephalic ectoderm primordium
GBrowse - Visual display of RNA-Seq signals
View Dmel\chif in GBrowse 22-52
Please Note FlyBase no longer curates genomic clone accessions so this list may not be complete
Please Note This section lists cDNAs and ESTs that fall within the genomic extent of the gene model, which may include cDNAs and ESTs of genes within introns, or of overlapping genes. Please see GBrowse for alignment of the cDNAs and ESTs to the gene model.
For each fully sequenced cDNA the DGRC maintains various forms of the cDNA (e.g tagged or untagged) in several different host vectors for subsequent cloning and expression in Drosophila and Drosophila cell lines.
Source for merge of: chif CG5817
Hypomorphic mutations in chif cause thin, fragile chorions and female sterility, and eliminate chorion gene amplification. Null alleles have the additional phenotypes of rough eyes and thin thoracic bristles; phenotypes often associated with disruption of the normal cell cycle.
chif is involved in processes in addition to chorion gene amplification.
Mutations at the chif locus cause defects in midoogenesis.
5 alleles are discussed but are not named.